What is Anti-SLC6A19 (Solute carrier family 6, member 19)?

SLC6A19, aswell accepted as B0AT1, is a affiliate of the Na+ -dependent neurotransmitter agent family, and mediates epithelial absorption of aloof amino acids beyond the aciculate film in the civil and the adjacent tubes of the renal cortex. Defective amino acerbic carriage due to the alteration of the SLC6A19 gene causes Hartnup disorder, which is characterized by a pellagra-like light-sensitive rash, cerebellar ataxia, affecting instability, and aminoaciduria.
This antibiotic can be acclimated to stain autogenous antigen in paraffin anchored animal tissues including branch and baby civil by Immunohistochemistry. The acuteness has been accepted by Western blotting, Immunocytochemistry and Flow cytometry to ascertain the abounding breadth of animal SLC6A19 briefly bidding in HEK 293T cells.
Solute carrier family 6 member 19 also known as the sodium-dependent neutral amino acid transporter B(0)AT1 or system B(0) neutral amino acid transporter AT1 is a protein that in humans is encoded by the SLC6A19 gene.
SLC6A19 is a system B(0) transporter that mediates epithelial resorption of neutral amino acids across the apical membrane in the kidney and intestine.
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